Myocarditis from the JABS -not covid- Here is the evidence I gathered. Counter the junk journalism! – Investment Watch


Okay, I’m literally *sick* of junk journalism continuously blaming covid (but never the jabs!) for myocarditis.
I even have put together a survey of the evidence showing (I imagine overwhelmingly) that the ‘vaccines’ –not covid– are in charge.
I actually don’t care if anyone desires to copy this, give it to Matt Le Tissier, or whatever!
This information must get on the market…

Journal of the American Medical Association
Myocarditis and Pericarditis After Vaccination for COVID-19
-> See p2 – charts.
-> Flat until 2021.
-> Not Covid.

Journal of the American Heart Association Circulation
Impact of Covid-19 on Acute Myocarditis Hospital Admissions within the National Health Service of England, Uk (2019-2020)
“Across all ages, there have been 1,894 hospital admissions as a result of myocarditis in 2019 compared with 1,610 in 2020 (15% reduction).”
“Throughout the first national lockdown (twenty third Mar-Nineteenth Jun 2020), myocarditis admissions were 32% lower than the identical period in 2019.”

-> Pre-vaccine (2020),
-> Large study, whole of England’s public hospitals.
-> No excess myocarditis, see Figure:

Journal of Clinical Medicine
The Incidence of Myocarditis and Pericarditis in Post COVID-19 Unvaccinated Patients-A Large Population-Based Study
No statistical difference within the incidence rate of each myocarditis (p =1) and pericarditis (p =0.17) was observed between the COVID-19 cohort and the control cohort
-> Paper written pre-vaccination.
-> Population n=4.4m people,

Journal of the American Medical Association Cardiology
SARS-CoV-2 Vaccination and Myocarditis in a Nordic Cohort Study of 23 Million Residents
-> 23 Million person study (Nordic contries).
-> A 10x to 32x risk over unvaccinated people of Myocarditis in under 40’s, when mixing Pfizer/Biontech (see table 2).

Journal Vaccines
Multifocal Necrotizing Encephalitis and Myocarditis after BNT162b2 mRNA Vaccination against COVID-19
“Since no nucleocapsid protein could possibly be detected, the presence of spike protein should be ascribed to vaccination moderately than to viral infection.”
“The findings corroborate previous reports of encephalitis and myocarditis brought on by gene-based COVID-19 vaccines.”

-> Critically, NO HISTORY OF COVID, no antibodies.
-> No Nucleocapsid from the virus found.
-> Spike is found at sites of inflammation (triple vaxxed).

Journal Molecular Science
Intramyocardial Inflammation after COVID-19 Vaccination: An Endomyocardial Biopsy-Proven Case Series
“Importantly, infectious causes have been excluded in all patients.”
“The expression of SARS-CoV-2 spike protein throughout the heart and the dominance of CD4+ lymphocytic infiltrates indicate an autoimmunological response to the vaccination.”
-> No covid infections.
-> Vaccine mechanism of motion and spike on the scene.

Okay, so it’s the vaccines… why?

#0 Spike is found on the ‘scene of the crime’ within the autopsy studies done by Arne Burkhardt & colleagues, yet the remaining of the viral proteins are missing.
#1 Mostly young people affected. Likely because their ribosomes are more efficient at translating the mRNA into spike than older people.
#2 Spike from vaccines goes in all places, even crossing the BBB as a result of the Lipid Nanoparticles.
#3 Spike from the vaccines is more durable. It uses N1-methylPseudoUridine, versus natural mRNA which is overwhelmingly just Uridine.

Journal Circulation
Circulating Spike Protein Detected in Post–COVID-19 mRNA Vaccine Myocarditis
-> See fig 4a.
-> It’s a LOG scale, too – (And the lower Limit of Detection is ’15’ pg/ml.)
-> Some patients have blood spike levels going *up* until they stopped measuring! (Thanks, N1-methylpseudouridine!)- see Fig. 4B.

Journal Clinical Infectious Diseases
Intravenous Injection of Coronavirus Disease 2019 (COVID-19) mRNA Vaccine Can Induce Acute Myopericarditis in Mouse Model

Preprint: cardiac disease in mice brought on by spike
The SARS-CoV-2 Spike protein induces long-term transcriptional perturbations of mitochondrial metabolic genes, causes cardiac fibrosis, and reduces myocardial contractile in obese mice

“Our data demonstrated that the Spike protein could induce long-term transcriptional suppression of mitochondria metabolic genes and cause cardiac fibrosis and myocardial contractile impairment, providing mechanistic insights to PACS-related cardiomyopathy.

h/t S-man

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